Author

S. T. Grey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:clinical cohort / trial· 1

Frequent Co-Authors

S. N. Walters· 2 shared A. E. Minoche· 1 shared A. Jakovija· 1 shared A. Kale· 1 shared A. O. Yam· 1 shared A. Russell· 1 shared C. Burk· 1 shared C. Counoupas· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersImmunology

S. N. Walters

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4 papers

Recent·Most cited

Novel metabolic role for CD47 in pancreatic β-cell insulin secretion and islet transplant outcomes

K. Ghimire, A. Kale, J. Li, S. M. Julovi, P. O'Connell, S. T. Grey, W. J. Hawthorne, J. E. Gunton, N. M. Rogers

This study identifies CD47 as a regulator of insulin secretion and islet transplant outcomes, showing that its inhibition enhances insulin release and transplant efficiency.

bioRxiv · Jul 22, 2022Physiology

Environmental and genetic disease modifiers of haploinsufficiency of A20

N. N. Zammit, P. Gray, O. M. Siggs, J. Y. Yap, A. Russell, D. Cultrone, J. Warren, S. N. Walters, R. T. Brink, D. Zahra, D. L. Burnett, V. Gayevskiy, A. E. Minoche, J. B. Ziegler, M. E. Craig, , S. T. Grey

The paper identifies genetic and environmental modifiers that exacerbate autoinflammatory disease caused by A20 haploinsufficiency in humans.

bioRxiv · Mar 19, 2022 · PreprintImmunology

Microbial activation converts neutrophils into anti-tumor effectors

A. O. Yam, J. Bailey, F. Lin, A. Jakovija, C. Counoupas, J. A. Triccas, M. Gunzer, T. Bald, S. T. Grey, T. Chtanova

This study shows microbial bioparticles convert neutrophils into anti-tumor effectors by altering their phenotype and function, leading to tumor growth inhibition.

bioRxiv · Aug 21, 2020 · PreprintImmunology

A20 is an immune tolerance factor that can determine islet transplantoutcomes.

N. W. Zammit, S. N. Walters, K. Seeberger, G. S. Korbutt, S. T. Grey

This study shows that increasing A20 expression in islet grafts improves allogeneic survival and reduces immune rejection, supporting the use of alternative islet sources with reduced immunosuppression.

bioRxiv · Sep 16, 2019Immunology