Author

S.-W. Kim

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Biophysics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

D. R. Williams· 2 shared D.-W. Jung· 2 shared B.-H. Oh· 1 shared B.-S. Jeong· 1 shared H. Kim· 1 shared H. Lee· 1 shared H.-J. Kim· 1 shared J. Song· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

D. R. Williams

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Targeting phosphatase DUSP22 ameliorates skeletal muscle wasting via Akt independent JNK-FOXO3a repression

D. R. Williams, S. H. LEE, H.-J. Kim, S.-w. Kim, H. Lee, D.-W. Jung

This study identifies DUSP22 as a novel target for preventing skeletal muscle wasting by repressing the JNK-FOXO3a pathway independently of Akt.

bioRxiv · Apr 8, 2024 · PreprintCell Biology

Noncovalent antibody catenation on a target surface drastically increases the antigen-binding avidity

J. Song, B.-S. Jeong, S.-W. Kim, S.-B. Im, W. Cho, M.-J. Ahn, B.-H. Oh

This study demonstrates that genetically fusing a weakly homodimeric protein to antibodies enhances antigen-binding avidity by up to 5,120-fold through reversible catenation on antigen-rich surfaces.

bioRxiv · Jul 12, 2022Biophysics

Inhibiting 5-lipoxygenase prevents skeletal muscle atrophy by targeting organogenesis signaling and insulin-like growth factor-1

H. Kim, S.-W. Kim, S.-H. Lee, D.-W. Jung, D. R. Williams

This study shows that inhibiting 5-lipoxygenase with malotilate prevents skeletal muscle atrophy by enhancing organogenesis signaling and insulin-like growth factor-1 expression.

bioRxiv · May 4, 2022 · PreprintCell Biology