Author

Sahar Sabry

National Research CentrePrimary ORCID profile (0000-0003-0607-6842 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Sahar SabryiD1 paper · Centre National de la Recherche Scientifique

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:metabolomics· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Akhilesh PandeyiD· 1 shared Alexia Tyler King· 1 shared Charlotte Bleukx· 1 shared Doughlas Y Smith· 1 shared Éva MoravaiD· 1 shared Graeme J. PrestoniD· 1 shared Ibrahim ShammasiD· 1 shared Jisha Chandran· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Akhilesh PandeyORCID

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Author profile →

1 paper

Recent·Most cited

ALG13 loss-of-function alters glycosylation, impairs neuronal maturation, and drives network hypoactivity in a cortical organoid model of CDG

Rameen Shah, Rohit Budhraja, Silvia Radenkovic, Graeme J. Preston, Alexia Tyler King, Sahar Sabry, Charlotte Bleukx, Ibrahim Shammas, Lyndsay E.A. Young, Jisha Chandran, Seul Kee Byeon, Ronald F. Hrstka, Doughlas Y Smith, Nathan P. Staff, Richard R. Drake, , Tamás Kozicz

This study uses cortical organoids to show that ALG13 loss-of-function causes glycosylation defects, neuronal maturation issues, and network hypoactivity in CDG.

bioRxiv · Jul 9, 2025 · PreprintNeuroscience