Author

Shiho Omori

Japan Anti Tuberculosis Association

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Hajime Nakamura· 3 shared Minako HijikataiD· 3 shared Naoto KeichoiD· 3 shared Shintaro SetoiD· 3 shared Haruka HikichiiD· 2 shared Koji FuruuchiiD· 2 shared Kozo MorimotoiD· 2 shared Michito HamadaiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

9 top collaborators · 32 mutual co-author links

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3 shared papersImmunology

Hajime Nakamura

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Clinical isolates of Mycobacterium avium complex reveal an M. intracellulare-associated IL-17/neutrophilic pulmonary immune program in a murine disease model

Shintaro Seto, Koji Furuuchi, Shiho Omori, Hajime Nakamura, Kozo Morimoto, Minako Hijikata, Naoto Keicho

The paper investigates immune responses to different Mycobacterium avium complex isolates in mice, finding that M. intracellulare induces a distinct IL-17/neutrophilic immune program compared to M. avium.

mSphere · Sep 21, 2026Peer-reviewedImmunology

Strain-dependent disease progression and necrotizing granuloma formation induced by virulent Mycobacterium avium complex strains in a murine model

Haruka Hikichi, Shiho Omori, Hajime Nakamura, Shintaro Seto, Koji Furuuchi, Kozo Morimoto, Minako Hijikata, Naoto Keicho

This study evaluates strain-dependent disease progression and necrotizing granuloma formation in a murine model of MAC-PD using five clinical MAC strains.

Microbiology Spectrum · Mar 24, 2026Peer-reviewedMicrobiology

Mafb Deficiency in Myeloid Cells Increases Susceptibility to Mycobacterium tuberculosis Infection in Mice

Haruka Hikichi, Hajime Nakamura, Shiho Omori, Shintaro Seto, Minako Hijikata, Michito Hamada, Satoru Takahashi, Naoto Keicho

This study shows that Mafb deficiency in myeloid cells increases susceptibility to Mycobacterium tuberculosis infection in mice by impairing macrophage function and immune cell recruitment.

Frontiers in Immunology · Jan 23, 2026Peer-reviewedImmunology