Author

Suey van Baarle

University of Alberta

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 1 Research Focus:spatial domain detection· 1 Approach:statistical model· 1 Organism:zebrafish· 1

Frequent Co-Authors

Andrew Jan WaskiewicziD· 2 shared Curtis R. FrenchiD· 2 shared Ordan J. LehmanniD· 2 shared Paul W. ChrystaliD· 2 shared Serhiy HavryloviD· 2 shared Ann-Marie PetursoniD· 1 shared David B. PilgrimiD· 1 shared Francesca JeaniD· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersGenetics

Andrew Jan WaskiewiczORCID

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2 papers

Recent·Most cited

The Axenfeld-Rieger syndrome gene FOXC1 contributes to left-right patterning

Paul W. Chrystal, Curtis R. French, Francesca Jean, Serhiy Havrylov, Suey van Baarle, Ann-Marie Peturson, Pengfei Xu, Justin Gage Crump, David B. Pilgrim, Ordan J. Lehmann, Andrew Jan Waskiewicz

This paper shows that FOXC1 mutations in zebrafish cause left-right axis defects and congenital heart defects, similar to human Axenfeld-Rieger syndrome.

Genes · Jan 26, 2021Peer-reviewedGenetics

Pleiotropy in FOXC1-attributable phenotypes involves altered ciliation and cilia-dependent signaling

Serhiy Havrylov, Paul W. Chrystal, Suey van Baarle, Curtis R. French, Ian M. MacDonald, Jagannadha Avasarala, R. Curtis Rogers, Fred B. Berry, Tsutomu Kume, Andrew Jan Waskiewicz, Ordan J. Lehmann

The paper shows that FOXC1 affects ciliation and cilia-dependent signaling, contributing to pleiotropic phenotypes in Axenfeld-Rieger Syndrome.

bioRxiv · Aug 13, 2020 · PreprintGenetics