Author

T. A. Harris-Tryon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Organism:mouse· 1

Frequent Co-Authors

M. Artami· 2 shared A. G. Lone· 1 shared A. R. Horswill· 1 shared C. C. Zouboulis· 1 shared C. Zhang· 1 shared J. G. A. McDonald· 1 shared J. Kavanaugh· 1 shared J. L. Pineider· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersMicrobiology

M. Artami

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Androgens at the skin surface regulate S. aureus pathogenesis through the activation of agr quorum sensing

M. S. John, M. Chinnappan, M. Artami, M. Bhattacharya, R. Keogh, J. Kavanaugh, T. Sharma, A. R. Horswill, T. A. Harris-Tryon

This study shows that androgens like testosterone promote S. aureus pathogenesis by activating the agr quorum sensing system, leading to increased virulence and skin infection.

bioRxiv · Feb 10, 2024 · PreprintMicrobiology

Characterization of cutaneous sex steroid hormone production through analysis of skin secretions

J. L. Pineider, K. Eckert, J. G. A. McDonald, T. A. Harris-Tryon

This study quantifies sex steroid hormones in skin secretions from different anatomical sites and sexes, finding site-specific and sex-based differences in hormone levels.

bioRxiv · Sep 24, 2021 · PreprintPhysiology

Small proline-rich proteins (SPRRs) are epidermally produced antimicrobial proteins that defend the cutaneous barrier by direct bacterial membrane disruption

C. Zhang, Z. Hu, A. G. Lone, M. Artami, M. Edwards, C. C. Zouboulis, M. Stein, T. A. Harris-Tryon

This study identifies SPRR1 and SPRR2 as antimicrobial proteins produced by sebocytes that defend the skin by disrupting bacterial membranes, showing increased susceptibility to infections in their absence.

bioRxiv · Sep 1, 2021Immunology