Author

T. A. Schmidt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

A. Struglics· 1 shared C. Jin· 1 shared D. Chan· 1 shared D. K. Brubaker· 1 shared E. A. Blaber· 1 shared G. D. Jay· 1 shared K. A. Thomsson· 1 shared L. I. Bjorkman· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPathology

A. Struglics

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2 papers

Recent·Most cited

Endogenous production of hyaluronan, PRG4, and cytokines is sensitive to cyclic loading in synoviocytes

M. Pendyala, P. S. Woods, D. K. Brubaker, E. A. Blaber, T. A. Schmidt, D. Chan

This study shows cyclic loading modulates hyaluronan, PRG4, and cytokine production in synoviocytes, with strain magnitude affecting gene expression and cytokine profiles.

bioRxiv · Apr 20, 2022Bioengineering

Cathepsin G Degrades Synovial Fluid Lubricin: Relevance For Osteoarthritis Pathogenesis

S. Huang, K. A. Thomsson, C. Jin, S. Alweddi, A. Struglics, O. Rolfson, L. I. Bjorkman, S. Kalamajsk, T. A. Schmidt, G. D. Jay, R. Krawetz, N. G. Karlsson, T. Eisler

This study shows that cathepsin G degrades lubricin in synovial fluid, contributing to osteoarthritis pathogenesis by reducing joint lubrication.

bioRxiv · Oct 15, 2019 · PreprintPathology