Author

T. Dlugos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Research Focus:metabolism & bioenergetics· 2 Assay:metabolomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

E. L. Goldberg· 2 shared I. Shchukina· 2 shared M. Artyomov· 2 shared V. D. Dixit· 2 shared Y.-H. Youm· 2 shared A. Letian· 1 shared A. Wang· 1 shared B. K. Hilliard· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersImmunology

E. L. Goldberg

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3 papers

Recent·Most cited

Innate immune cell-intrinsic ketogenesis is dispensable for organismal metabolism and age-related inflammation

E. L. Goldberg, A. Letian, T. Dlugos, C. Leveau, V. D. L. Dixit

This study shows that ketogenesis in innate immune cells is not essential for metabolism or age-related inflammation, but liver-derived ketone bodies are critical for controlling inflammation and metabolism during aging.

bioRxiv · Oct 14, 2022Immunology

Ketogenesis restrains aging-induced exacerbation of COVID in a mouse model

S. Ryu, I. Shchukina, Y.-H. Youm, H. Qing, B. K. Hilliard, T. Dlugos, X. Zhang, Y. Yasumoto, C. J. Booth, C. Fernandez-Hernando, Y. Suarez, K. M. Khanna, T. Horvath, M. O. Dietrich, M. Artyomov, , V. D. Dixit

This study shows that a ketogenic diet reduces inflammation and improves outcomes in aged mice infected with a coronavirus, suggesting potential therapeutic benefits for elderly COVID-19 patients.

bioRxiv · Sep 11, 2020 · PreprintImmunology

Dysregulation of adipose ILC2 underlies thermogenic failure in aging

E. L. Goldberg, I. Shchukina, Y.-H. Youm, C. D. Camell, T. Dlugos, M. Artyomov, V. D. Dixit

The paper identifies age-related changes in adipose tissue that reprogram ILC2, leading to impaired cold tolerance in aged mice.

bioRxiv · Sep 8, 2020 · PreprintImmunology