Author

T. Köcher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Plant Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:plant (other)· 1

Frequent Co-Authors

C. Bonnot· 2 shared C. Champion· 2 shared L. Dolan· 2 shared A. Barros· 1 shared A. Casey· 1 shared A. Neves-Costa· 1 shared C. Casey· 1 shared D. Pedroso· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersPlant Biology

C. Bonnot

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Reduced coenzyme Q synthesis confers non-target site resistance to the herbicide thaxtomin A

C. Casey, T. Köcher, C. Champion, K. Jandrasits, M. Mosiolek, C. Bonnot, L. Dolan

This study identifies loss-of-function mutations in MpRTN4IP1L as a novel mechanism of non-target site resistance to the herbicide thaxtomin A in Marchantia polymorpha.

bioRxiv · Sep 13, 2022Plant Biology

Transcriptome changes in chlorsulfuron-treated plants are caused by acetolactate synthase inhibition and not induction of a herbicide detoxification system in Marchantia polymorpha

A. Casey, T. Köcher, S. Caygill, C. Champion, C. Bonnot, L. Dolan

This study shows that chlorsulfuron-induced transcriptome changes in Marchantia polymorpha are due to acetolactate synthase inhibition, not a detoxification response.

bioRxiv · Aug 31, 2022Plant Biology

Host-dependent induction of disease tolerance to infection by tetracycline antibiotics

H. G. Colaco, A. Barros, A. Neves-Costa, E. Seixas, D. Pedroso, T. R. Velho, K. Willmann, H.-S. Yi, M. Shong, V. Benes, S. Weis, T. Köcher, L. F. Moita

This study shows tetracycline antibiotics protect against sepsis by inducing disease tolerance through mitochondrial protein synthesis inhibition, independent of their direct antibiotic effects.

bioRxiv · Nov 7, 2019Immunology