Author

T. Lasho

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:whole-exome sequencing· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

J. Zhang· 2 shared M. M. Patnaik· 2 shared A. A. Mangaonkar· 1 shared A. Buradkar· 1 shared A. Rajagopalan· 1 shared A. Vedder· 1 shared A. Veltri· 1 shared A. Yurchenko· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

J. Zhang

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Author profile →

2 papers

Recent·Most cited

Genomic and transcriptional profiling stratifies VQ myeloma lines into two clusters with distinct risk signatures and drug responses

E. Flietner, M. Yu, A. Rajagopalan, Y. Zhou, Y. Feng, A. Veltri, T. Lasho, Z. Wen, Y. Sun, M. M. Patnaik, N. S. Callander, F. Asimakopoulos, D. Wang, J. Zhang

bioRxiv · Aug 21, 2022 · PreprintCancer Biology

RAS mutations drive proliferative chronic myelomonocytic leukemia via activation of a novel KMT2A-PLK1 axis

R. M. Carr, D. Vorobyev, T. Lasho, D. L. Marks, E. J. Tolosa, A. Vedder, L. Almada, A. Yurchenko, I. Padioleau, B. Alver, G. Coltro, M. Binder, S. L. Safgren, I. Horn, X. You, , M. M. Patnaik

This study identifies RAS mutations as drivers of proliferative CMML through a novel KMT2A-PLK1 axis, showing PLK1 inhibition can improve disease outcomes in patient-derived xenografts.

bioRxiv · Dec 23, 2019 · PreprintCancer Biology