Author

V. N. Fritsch

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

H. Antelmann· 2 shared A. J. Slusarenko· 1 shared C. Hamilton· 1 shared D. Niemeyer· 1 shared J. Bernhardt· 1 shared K. Mösbauer· 1 shared K. Tedin· 1 shared L. Adrian· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersMicrobiology

H. Antelmann

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2 papers

Recent·Most cited

Allicin inhibits SARS-CoV-2 replication and abrogates the antiviral host response in the Calu-3 proteome

K. Mösbauer, V. N. Fritsch, L. Adrian, J. Bernhardt, M. C. H. Gruhlke, A. J. Slusarenko, D. Niemeyer, H. Antelmann

This study shows allicin inhibits SARS-CoV-2 replication and suppresses the antiviral host response in Calu-3 cells.

bioRxiv · May 15, 2021 · PreprintMicrobiology

Staphylococcus aureus uses the bacilliredoxin (BrxAB)/bacillithiol disulfide reductase (YpdA) redox pathway to defend against oxidative stress under infections

N. Linzner, V. V. Loi, V. N. Fritsch, Q. N. Tung, S. Stenzel, M. Wirtz, R. Hell, C. Hamilton, K. Tedin, M. Fulde, H. Antelmann

This study identifies the BrxAB/BSH/YpdA pathway as critical for S. aureus to manage oxidative stress and survive infections by maintaining BSH redox homeostasis.

bioRxiv · May 1, 2019Microbiology