Author

V. Studentsova

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Bioengineering· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Research Focus:development & morphogenesis· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cancer & tumor microenvironment· 1 Approach:genetic perturbation / CRISPR· 1 Assay:imaging· 1

Frequent Co-Authors

A. Loiselle· 4 shared E. Knapp· 2 shared J. Ackerman· 2 shared K. Best· 1 shared K. Mora· 1 shared M. Buckley· 1 shared M. Glasner· 1 shared

Activity by Year

2017-2019 · peak 2/yr

Co-Author Collaboration Network

7 top collaborators · 12 mutual co-author links

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4 shared papersCell Biology

A. Loiselle

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cell non-autonomous functions of S100a4 drive fibrotic tendon healing

J. Ackerman, V. Studentsova, K. Best, E. Knapp, A. Loiselle

This study shows that inhibiting S100a4 improves tendon healing by reducing scar formation and enhancing mechanical properties through cell non-autonomous mechanisms.

bioRxiv · Jan 9, 2019Cell Biology

Non-invasive ultrasound quantification of Scar Tissue Volume predicts functional changes during tendon healing

J. Ackerman, V. Studentsova, A. Loiselle

This study quantifies scar tissue volume via ultrasound to predict functional changes during tendon healing and identifies its utility in screening therapeutic interventions.

bioRxiv · Nov 4, 2018Bioengineering

Insulin Receptor Deletion in S100a4-lineage cells accelerates age-related bone loss

V. Studentsova, E. Knapp, A. Loiselle

This study deletes the insulin receptor in S100a4-lineage cells in mice and finds accelerated age-related bone loss in older adults.

bioRxiv · Oct 16, 2018Cell Biology

Obesity/Type II Diabetes Promotes Function-Limiting Changes In Flexor Tendon Extracellular Matrix Organization That Are Not Reversed By Restoring Normal Metabolic Function

V. Studentsova, K. Mora, M. Glasner, M. Buckley, A. Loiselle

This study develops a murine model of diabetic tendinopathy and shows that restoring normal metabolic function does not reverse tendon impairments caused by obesity/T2DM.

bioRxiv · Dec 12, 2017Pathology