Author

V. V. Costa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1

Frequent Co-Authors

C. M. Queiroz-Junior· 3 shared D. G. Souza· 2 shared T. P. Moreira· 2 shared A. A. Santos· 1 shared A. Bafica· 1 shared A. C. Santos· 1 shared A. Morrot· 1 shared B. R. Barrioni· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersCell Biology

C. M. Queiroz-Junior

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Essential role of the CCL2-CCR2 axis in Mayaro virus-induced disease

F. M. Santos, V. C. Melo, S. Araujo, C. D. F. Sousa, T. P. Moreira, M. R. Goncalves, A. C. Santos, H. A. Seabra, P. A. C. Costa, B. R. Barrioni, P. B. Souza, M. d. M. Pereira, M. L. Nogueira, D. G. Souza, P. P. Guimaraes, , V. V. Costa

This study shows that the CCL2-CCR2 axis drives inflammation and bone loss in Mayaro virus infection by recruiting macrophages and promoting osteoclastogenesis.

bioRxiv · Jul 21, 2023Cell Biology

Targeting the Annexin A1-FPR2/ALX pathway for host-directed therapy in dengue disease

V. V. Costa, M. A. Sugimoto, J. Hubner, C. S. Bonilha, C. M. Queiroz-Junior, M. H. Goncalves-Pereira, J. Chen, T. Gobbetti, G. O. Libanio Rodrigues, J. L. Bambirra, I. B. Passos, C. E. Machado Lopes, T. P. Moreira, K. Bonjour, R. C. N. Melo, , M. M. Teixeira

This study identifies reduced Annexin A1 levels in dengue patients and mice, showing that AnxA1 agonists reduce disease severity by modulating mast cell degranulation.

bioRxiv · Nov 2, 2021Microbiology

Neuraminidase inhibitors rewire neutrophil function in vivo in murine sepsis and ex vivo in COVID-19

R. O. Formiga, F. C. Amaral, C. F. Souza, D. A. G. B. Mendes, C. W. S. Wanderley, C. B. Lorenzini, A. A. Santos, J. Antonia, L. F. Faria, C. C. Natale, N. M. Paula, P. C. S. Silva, F. R. Fonseca, L. Aires, N. Heck, , F. Spiller

This paper shows neuraminidase inhibitors reduce neutrophil overactivation in sepsis and COVID-19 by inhibiting sialic acid release and ROS production.

bioRxiv · Nov 12, 2020 · PreprintImmunology