Author

W. Salgado-Pabon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 5

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 5 Approach:experimental assay· 4 Organism:human· 3 Organism:rat· 2 Assay:bulk RNA-seq· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

K. J. Kinney· 3 shared K. Kulhankova· 2 shared P. M. Tran· 2 shared S. S. Tang· 2 shared A. N. Forsythe· 1 shared B. P. Howden· 1 shared C. Bouchiat· 1 shared E. M. Garcia· 1 shared

Activity by Year

2019-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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3 shared papersMicrobiology

K. J. Kinney

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

IL-17C is a driver of damaging inflammation during Neisseria gonorrhoeae infection of human Fallopian tube

E. M. Garcia, J. D. Lenz, K. T. Hackett, W. Salgado-Pabon, J. P. Dillard

This study identifies IL-17C as a key driver of inflammation and tissue damage in human Fallopian tubes during Neisseria gonorrhoeae infection.

bioRxiv · Nov 28, 2022 · PreprintMicrobiology

All Staphylococcus aureus bacteraemia strains have the potential to cause infective endocarditis: results of GWAS and experimental animal studies

S. Bastien, S. Meyers, W. Salgado-Pabon, S. Giulieri, J.-P. Rasigade, L. Liesenborghs, K. J. Kinney, F. Couzon, P. Martin-Simoes, V. Le Moing, X. M. Duval, N. E. Holmes, N. E. Bruun, R. Skov, B. P. Howden, , F. Vandenesch

This study shows that all Staphylococcus aureus bacteraemia strains can cause infective endocarditis, as no genetic differences were found between IE and non-IE strains using GWAS and experimental models.

bioRxiv · May 16, 2022 · PreprintMicrobiology

Vegetation formation in Staphylococcus aureus endocarditis inversely correlates with RNAIII and sarA expression in invasive clonal complex 5 (CC5) isolates

K. J. Kinney, J. M. Stach, K. Kulhankova, M. Brown, W. Salgado-Pabon

bioRxiv · Apr 20, 2022 · PreprintMicrobiology

Staphylococcus aureus β-toxin exerts anti-angiogenic effects by inhibiting re-endothelialization and neovessel formation

P. M. Tran, S. S. Tang, W. Salgado-Pabon

This study shows that S. aureus β-toxin inhibits endothelial cell proliferation and migration, reducing angiogenesis and wound repair, which supports bacterial infection.

bioRxiv · Nov 26, 2021Microbiology

SEC is an anti-angiogenic virulence factor that promotes Staphylococcus aureus Infective Endocarditis Independent of Superantigen Activity

K. J. Kinney, S. S. Tang, X.-J. Wu, P. M. Tran, N. Bharadwaj, K. N. Gibson-Corley, A. N. Forsythe, K. Kulhankova, J. Gumperz, W. Salgado-Pabon

This study shows SEC promotes SAIE through anti-angiogenic effects, independent of its superantigen activity.

bioRxiv · Dec 13, 2019 · PreprintMicrobiology