Author

J. P. Dillard

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 8

Signature Assays, Organisms & Methods

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Approach:experimental assay· 7 Organism:human· 7 Assay:proteomics· 4 Research Focus:cell type annotation· 3 Research Focus:host-pathogen & immune response· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:differential expression· 2

Frequent Co-Authors

K. T. Hackett· 4 shared R. E. Schaub· 4 shared K. M. Perez-Medina· 3 shared A. K. Klimowicz· 2 shared E. M. Garcia· 2 shared J. M. Chan· 2 shared A. C. Shockey· 1 shared C. Pepperell· 1 shared

Activity by Year

2021-2024 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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4 shared papersMicrobiology

K. T. Hackett

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Author profile →

8 papers

Recent·Most cited

Peptidoglycan fragment release and NOD activation by commensal Neisseria species from humans and other animals

T. N. Harris-Jones, J. M. Chan, K. T. Hackett, N. J. Weyand, R. E. Schaub, J. P. Dillard

This study examines PG fragment release and NOD activation in nonpathogenic Neisseria species from humans and animals, finding species-specific differences in PG release and NOD responses.

bioRxiv · Jan 9, 2024 · PreprintMicrobiology

Mutation of mltG increases peptidoglycan fragment release, cell size, and antibiotic susceptibility in Neisseria gonorrhoeae

T. N. Harris, K. M. Perez-Medina, K. T. Hackett, M. A. Schave, R. E. Schaub, J. P. Dillard

bioRxiv · Aug 23, 2023 · PreprintMicrobiology

Mutational analysis of LtgC, a lytic transglycosylase required for cell separation in Neisseria gonorrhoeae

R. E. Schaub, K. M. Perez-Medina, J. Tomberg, R. A. Nicholas, J. P. Dillard

This study investigates the role of LtgC in cell separation and peptidoglycan degradation in Neisseria gonorrhoeae by analyzing mutants with amino acid substitutions and domain deletions.

bioRxiv · Jun 20, 2023 · PreprintMicrobiology

Genotypic and phenotypic differences among phase-variable colony variants conserved across Gardnerella spp.

E. M. Garcia, A. K. Klimowicz, L. Edupuganti, M. A. Topf, S. R. Bhide, D. J. Slusser, S. M. Leib, G. A. Buck, K. K. Jefferson, C. S. Pepperell, J. P. Dillard

This study identifies phase-variable colony variants in Gardnerella species and their potential roles in bacterial vaginosis pathogenesis.

bioRxiv · Jan 13, 2023 · PreprintMicrobiology

IL-17C is a driver of damaging inflammation during Neisseria gonorrhoeae infection of human Fallopian tube

E. M. Garcia, J. D. Lenz, K. T. Hackett, W. Salgado-Pabon, J. P. Dillard

This study identifies IL-17C as a key driver of inflammation and tissue damage in human Fallopian tubes during Neisseria gonorrhoeae infection.

bioRxiv · Nov 28, 2022 · PreprintMicrobiology

Listeria monocytogenes GlmR is an accessory uridyltransferase essential for cytosolic survival and virulence

D. Pensinger, K. V. Gutierrez, H. B. Smith, W. J. B. Vincent, D. M. Stevenson, K. A. Black, K. M. Perez-Medina, J. P. Dillard, K. Y. Rhee, D. Amador-Noguez, T. N. Huynh, J.-D. Sauer

This study identifies GlmR as an accessory uridyltransferase essential for Listeria monocytogenes cytosolic survival and virulence by synthesizing UDP-GlcNAc.

bioRxiv · Oct 27, 2021 · PreprintMicrobiology

The AmiC/NlpD pathway dominates peptidoglycan breakdown in Neisseria meningitidisand affects cell separation, NOD1 agonist production, and infection

J. M. Chan, K. T. Hackett, K. L. Woodhams, R. E. Schaub, J. P. Dillard

This study shows that the AmiC/NlpD pathway in N. meningitidis produces PG-derived peptides that activate NOD1 and are essential for cell separation and infection.

bioRxiv · Sep 2, 2021Microbiology

Transcriptional and translational responsiveness of the Neisseria gonorrhoeae type IV secretion system to conditions of host infections

M. M. Callaghan, A. K. Klimowicz, A. C. Shockey, J. Kane, C. Pepperell, J. P. Dillard

The paper identifies conditions that increase Neisseria gonorrhoeae type IV secretion system expression through transcriptional and translational mechanisms, and shows upregulation of these genes during urethral infection.

bioRxiv · Feb 12, 2021Microbiology