Author

W.-M. Deng

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 3 Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Tian· 2 shared Y.-C. Huang· 2 shared C. A. M. Costa· 1 shared D. Chatterjee· 1 shared D. Tian· 1 shared F. Cong· 1 shared H. Liu· 1 shared J. Jiang· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCell Biology

A. Tian

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Topoisomerase I is an Evolutionarily Conserved Key Regulator for Satellite DNA Transcription

H. Liu, Z. Teng, L. Yang, Q. Zhang, Y. Chen, X. Wang, Y. Zheng, A. Tian, D. Tian, Z. Lin, W.-M. Deng

This study identifies Topoisomerase I as a conserved regulator of satellite DNA transcription across species, showing its role in centromere function and DNA damage response.

bioRxiv · May 3, 2024Cell Biology

Cell polarity opposes Jak-STAT mediated Escargot activation that drives intratumor heterogeneity in a Drosophila tumor model

D. Chatterjee, F. Cong, X.-F. Wang, C. A. M. Costa, Y.-C. Huang, W.-M. Deng

The paper shows that cell polarity prevents Jak-STAT signaling from activating Escargot, reducing intratumor heterogeneity in a Drosophila tumor model.

bioRxiv · Dec 12, 2022 · PreprintCancer Biology

Re-entry into mitosis and regeneration of intestinal stem cells through enteroblast dedifferentiation in Drosophila midguts

A. Tian, V. Morejon, S. Kohoutek, Y.-C. Huang, W.-M. Deng, J. Jiang

This study shows that enteroblasts in Drosophila midguts re-enter mitosis and dedifferentiate into intestinal stem cells via EGFR-Ras signaling, revealing a mechanism for tissue regeneration.

bioRxiv · Nov 22, 2021 · PreprintCell Biology