Author

Ron R. Kopito

Stanford UniversityORCID 0000-0001-5027-6935Unclaimed ORCID profile · follow to get notified of new preprints

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Ron R. Kopito1 paper

Biology Disciplines

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Cell Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:Drosophila· 1 Assay:proteomics· 1

Frequent Co-Authors

Alexandra PanoviD· 1 shared Aprem Zaya· 1 shared Arda MizrakiD· 1 shared Enya Miguel WhelaniD· 1 shared Gabrielle E Pisano· 1 shared J. Wade HarperiD· 1 shared Kelsey L. HickeyiD· 1 shared Kirby M. DonnellyiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCell Biology

Alexandra PanovORCID

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2 papers

Recent·Most cited

Cell BiologyProceedings of the National Academy of SciencesPeer-reviewedDec 5, 2024

Temporal control of acute protein aggregate turnover by UBE3C and NRF1-dependent proteasomal pathways

Kelsey L. Hickey, Alexandra Panov, Enya Miguel Whelan…·J. Wade Harper

This study identifies UBE3C and NRF1-dependent proteasomal pathways that regulate acute protein aggregate turnover in response to misfolded protein accumulation.

8 citations

proteomicsexperimental assay

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NeuroscienceeLifePeer-reviewedMay 28, 2020

Phagocytic glia are obligatory intermediates in transmission of mutant huntingtin aggregates across neuronal synapses

Kirby M. Donnelly, Olivia R DeLorenzo, Aprem Zaya…·Margaret M.P. Pearce

The paper shows that mutant huntingtin aggregates spread between neurons via phagocytic glia in Drosophila, requiring the Draper receptor and synaptic dysfunction.

37 citations

Drosophilaexperimental assay

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