Author

Anne Hamacher‐Brady

Johns Hopkins UniversityORCID 0000-0001-5042-1338Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Anthony K. L. LeungiD· 1 shared Arianna Franca AnzmanniD· 1 shared Banhi BiswasiD· 1 shared Che-Yuan Chang· 1 shared Chenyao WangiD· 1 shared Danfeng CaiiD· 1 shared Deepti Pai Tonse· 1 shared Grant J. ButschekiD· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCell Biology

Anthony K. L. LeungORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintAug 18, 2025

The Cancer/Testis Antigen FATE1 Antagonizes Fission and Preserves Mitochondrial Network Integrity under Cytotoxic Stress

Verena Lang, Jessica Zahn, Deepti Pai Tonse…·Anne Hamacher‐Brady

This study identifies FATE1 as a mitochondrial fusion-promoting factor that antagonizes fission and protects against fragmentation under cytotoxic stress.

DOI 10.1101/2025.08.18.670752

humanproteomics

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Cell BiologyThe EMBO JournalPeer-reviewedApr 7, 2025

Interferon-Induced PARP14-Mediated ADP-Ribosylation in p62 Bodies Requires an Active Ubiquitin-Proteasome System

Rameez Raja, Banhi Biswas, Rachy Abraham…·Anthony K. L. Leung

This study shows interferon induces ADP-ribosylation in p62 bodies via PARP14, requiring ubiquitin activation and proteasome activity.

18 citationstop 3%

humanproteomics

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Cell BiologyHuman Molecular GeneticsPeer-reviewedOct 29, 2024

Stem cell models of TAFAZZIN deficiency reveal novel tissue-specific pathologies in Barth Syndrome

Olivia Sniezek Carney, Kodi William Harris, Yvonne Wohlfarter…·Hilary J. Vernon

This study uses stem cell models to investigate tissue-specific pathologies in Barth Syndrome caused by TAFAZZIN deficiency, identifying mitophagy defects and potential therapeutic targets.

9 citations

humanscRNA-seq

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