Author

Xander H.T. Wehrens

Baylor College of MedicineORCID 0000-0001-5044-672XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:differential expression· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Yuriana Aguilar-SáncheziD· 2 shared Adam G. Rokita· 1 shared Anahita AboonabiiD· 1 shared Andrielle Elaine CapoteiD· 1 shared Anna MalovannayaiD· 1 shared Anthony Tucker-BartleyiD· 1 shared Antrix JainiD· 1 shared Arvind SridhariD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersSystems Biology

Yuriana Aguilar-SánchezORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Systems BiologyJournal of Molecular and Cellular CardiologyPeer-reviewedMay 1, 2024

Atrial Proteomic Profiling Reveals a Switch Towards Profibrotic Gene Expression Program in CREM-IbΔC-X Mice with Persistent Atrial Fibrillation

Shuai Zhao, Mohit M. Hulsurkar, Satadru K. Lahiri…·Xander H.T. Wehrens

This study identifies proteomic changes in CREM-IbΔC-X transgenic mice with persistent atrial fibrillation, showing a shift from metabolic to profibrotic gene expression programs with age.

5 citations

mouseproteomics

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Molecular BiologybioRxivPreprintApr 19, 2023

Myosin Light Chain Dephosphorylation by PPP1R12C Promotes Atrial Hypocontractility in Atrial Fibrillation

Srikanth Perike, Francisco J. González-González, Issam Abu-Taha…·Mark D. McCauley

This study shows PPP1R12C overexpression in AF patients and mice leads to MLC2a dephosphorylation, reduced atrial contractility, and increased AF inducibility.

2 citations

humanmouse

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Molecular BiologybioRxivPreprintFeb 18, 2020

O-GlcNAcylation, oxidation and CaMKII contribute to atrial fibrillation in type 1 and type 2 diabetes by distinct mechanisms

Olurotimi O. Mesubi, Adam G. Rokita, Neha Abrol…·Mark E. Anderson

This study shows that O-GlcNAcylation and oxidative stress via CaMKII contribute to atrial fibrillation in type 1 and type 2 diabetes through distinct mechanisms.

1 citation

mouseexperimental assay

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