Author

Haining Zhu

University of ArizonaORCID 0000-0001-5498-6694Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Chunlong MaiD· 2 shared Alma L. BurlingameiD· 1 shared Anika Tapita· 1 shared Annie LiiD· 1 shared Avijite Kumer SarkariD· 1 shared Cenjing Wang· 1 shared Christina GrossiD· 1 shared Courtney N. BuchananiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Chunlong MaORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintOct 28, 2025

Acetylation of Axonal G3BP1 through ELP3 Accelerates Axon Regeneration

Irene Dalla Costa, Emily Michenfelder, Sophia Siciliano…·Jeffery L. Twiss

This study shows that acetylation of G3BP1 by ELP3 promotes axon regeneration and neuronal recovery after injury.

DOI 10.1101/2025.10.28.685144

mouseproteomics

𝕏 PostBluesky

NeurosciencebioRxivPreprintOct 14, 2024

An iPSC model of fragile X syndrome reflects clinical phenotypes and reveals m6A-mediated epi-transcriptomic dysregulation underlying synaptic dysfunction

Lu Lu, Avijite Kumer Sarkar, Lan Dao…·Ziyuan Guo

This study uses iPSC-derived neurons to show that FMRP deficiency in fragile X syndrome leads to m6A-mediated transcriptome dysregulation, causing synaptic dysfunction and suggesting therapeutic targets.

DOI 10.1101/2024.10.14.618205

𝕏 PostBluesky