Author

Avijite Kumer Sarkar

Cincinnati Children's Hospital Medical CenterORCID 0000-0002-7646-6566Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:differential expression· 1 Assay:electronic health records· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Annie LiiD· 1 shared Cenjing Wang· 1 shared Christina GrossiD· 1 shared Chunlong MaiD· 1 shared Crace Westerkamp· 1 shared Craig A. EricksoniD· 1 shared Dan LiuiD· 1 shared Ernest V. PedapatiiD· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Annie LiORCID

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2 papers

Recent·Most cited

An iPSC model of fragile X syndrome reflects clinical phenotypes and reveals m6A-mediated epi-transcriptomic dysregulation underlying synaptic dysfunction

Lu Lu, Avijite Kumer Sarkar, Lan Dao, Yanchen Liu, Chunlong Ma, Phyo Han Thwin, Xuyao Chang, George Yoshida, Annie Li, Cenjing Wang, Crace Westerkamp, Lauren Schmitt, Maag Chelsey, Monzon Stephanie, Yu Zhao, , Ziyuan Guo

This study uses iPSC-derived neurons to show that FMRP deficiency in fragile X syndrome leads to m6A-mediated transcriptome dysregulation, causing synaptic dysfunction and suggesting therapeutic targets.

bioRxiv · Oct 14, 2024 · PreprintNeuroscience

Electrophysiological maturation and increased excitability of human iPSC-derived neurons in HTR2A variant-related sleep bruxism

Avijite Kumer Sarkar, Shiro Nakamura, Kento Nakai, Takahiro Shiga, Yuka Abe, Yurie Hoashi, Tomio Inoue, Wado Akamatsu, Kazuyoshi Baba

This study examines electrophysiological differences in iPSC-derived neurons from sleep bruxism patients with a HTR2A variant, finding altered action potential properties compared to controls.

bioRxiv · Jan 26, 2021 · PreprintNeuroscience