Author

Miriam Lee

Dartmouth CollegeORCID 0000-0001-5500-1201Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Henry N. HiggsiD· 2 shared Aiman JalmukhambetovaiD· 1 shared Balajikarthick SubramanianiD· 1 shared Cristian V. RiellaiD· 1 shared Marie-Flore HenninoiD· 1 shared Martin R. PollakiD· 1 shared Sarah WilliamsiD· 1 shared Seth L. AlperiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCell Biology

Henry N. HiggsORCID

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2 papers

Recent·Most cited

Cell BiologyThe Journal of Cell BiologyPeer-reviewedNov 6, 2025

Regulation of the formin INF2 by actin monomers and calcium-calmodulin

Miriam Lee, Aiman Jalmukhambetova, Tucker Burgin, Henry N. Higgs

This study investigates INF2 regulation by calcium-calmodulin and actin monomers, showing that actin monomer-binding proteins are essential for INF2 activation and regulation.

7 citations

experimental assay

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GeneticsbioRxivPreprintJun 8, 2024

Missense Mutant Gain-of-Function Causes Inverted Formin 2 (INF2)-Related Focal Segmental Glomerulosclerosis (FSGS)

Balajikarthick Subramanian, Sarah Williams, Sophie Karp…·Martin R. Pollak

This study shows that a gain-of-function INF2 mutation causes FSGS in mice, linking INF2's cellular roles to disease progression and explaining its autosomal dominant inheritance.

4 citations

mouseproteomics

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