Pamela Stanley

The paper shows that absence of EOGT prevents developmental defects in Fringe-null mouse intestine, highlighting EOGT's role in intestinal development.

This study identifies DHRSX gene absence as the cause of dolichol synthesis defect in Lec5 and Lec9 CHO cells, correcting N-glycosylation defects through DHRSX complementation.

The paper shows that EOGT and O-GlcNAc glycans provide residual Notch signaling, enabling viability in mice lacking Pofut1 in the intestine.

This study identifies MGAT4D as a protective factor for male germ cells against mild heat stress, showing that its absence increases sensitivity to heat.