Author

Jingxuan Chen

The University of Texas Southwestern Medical CenterORCID 0000-0001-5931-0239Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:genetic perturbation / CRISPR· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

Chuanhai ZhangiD· 2 shared Dengbao YangiD· 2 shared Hao ZhuiD· 2 shared Hiroyuki SuzukiiD· 2 shared Jeon LeeiD· 2 shared Jingjing WangiD· 2 shared Mei-Jung Lin· 2 shared Meijuan BaiiD· 2 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

Chuanhai ZhangORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJan 11, 2026

Interorganelle competition for linoleic acid underlies steatotic liver pathology

Chuanhai Zhang, Dengbao Yang, Hiroyuki Suzuki…·Xing Zeng

This study identifies CLSTN3B as a driver of MASLD by promoting LD biogenesis and altering lipid partitioning between LDs and mitochondria, leading to mitochondrial dysfunction and oxidative stress.

DOI 10.64898/2026.01.11.698890

mousehuman

𝕏 PostBluesky

PhysiologybioRxivPreprintMay 20, 2025

Hepatocyte-specific CLSTN3B ablation impairs lipid droplet maturation and alleviates diet-induced steatohepatitis in mice

Chuanhai Zhang, Dengbao Yang, Hiroyuki Suzuki…·Xing Zeng

This study shows that CLSTN3B promotes lipid droplet maturation in hepatocytes and that its deletion reduces steatohepatitis in mice, with a positive correlation between CLSTN3B expression and MASLD severity in humans.

DOI 10.1101/2025.05.20.655199

mousehuman

𝕏 PostBluesky