Andres Gabriel Vidal-Gadea

The paper shows that dystrophic muscle has an elevated precontractile calcium set point sustained by limited PMCA1 export in C. elegans and human myoblasts.

This study shows that C. elegans muscle structure changes based on locomotion type, with swimmers having shorter, denser sarcomeres and elongated muscles compared to crawlers.

This study independently confirms that C. elegans can transmit learned PA14 avoidance to F1 and F2 generations, contradicting a recent study that failed to observe this beyond F1.

This study shows that fibrillin-related proteins regulate calcium homeostasis in dystrophic muscle across species, linking ECM integrity to calcium control.

This study investigates the role of PEZO-1 in C. elegans body wall muscles, showing it differentially affects crawling and swimming by modulating calcium dynamics and muscle activation.

This study identifies the role of AMsh glia in modulating the duration of nose-touch escape responses in C. elegans by mechanosensory transduction and GABAergic signaling.

The paper shows that Ih-induced membrane potential depolarization maintains the dynamic range of voltage-dependent gap junction signaling in electrical synapses.

This study identifies distinct pezo-1 isoforms in C. elegans with tissue-specific expression and functional roles in food intake regulation.

This study shows that sarcoplasmic calcium dysregulation occurs before structural muscle damage in C. elegans modeling Duchenne muscular dystrophy and that increased activity can improve some muscular parameters but not longevity.

The paper reanalyzes conflicting data on C. elegans magnetotaxis, highlights methodological differences, and presents new data showing how temporal and spatial factors influence magnetotaxis behavior.