Takahisa Maki

The paper identifies two motifs in the Swi2-Swi5 complex that enable cell-type-specific localization to recombination enhancers and stimulate Rad51-driven gene conversion in fission yeast.

This study identifies HULC and Set1C as factors that regulate heterochromatin organization during mating-type switching in fission yeast by controlling recombination enhancer usage and histone modifications.

This study reveals the roles of Rad51's DNA binding motifs in the catalytic process of DNA strand exchange during homologous recombination.

The paper identifies two binding sites on Sfr1 that interact with Rad51 and shows that Swi5-Sfr1 requires Rad55-Rad57 for DNA repair.