Author

Benjamin Steyer

University of Wisconsin–MadisonORCID 0000-0001-6002-511XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Alireza Fotuhi SiahpiraniiD· 1 shared Amr A. AbdeeniD· 1 shared Bikash Ranjan PattnaikiD· 1 shared Budd A. TuckeriD· 1 shared Cole Bacig· 1 shared David M. GammiD· 1 shared Divya SinhaiD· 1 shared Edwin M. StoneiD· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperGenetics

Alireza Fotuhi SiahpiraniORCID

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1 paper

Recent·Most cited

Human iPSC modeling reveals mutation-specific responses to gene therapy in Best disease

Divya Sinha, Benjamin Steyer, Pawan Kumar Shahi, Katherine P. Mueller, Rasa Valiauga, Kimberly L. Edwards, Cole Bacig, Stephanie S. Steltzer, Sandhya Srinivasan, Amr A. Abdeen, Evan Cory, Viswesh Periyasamy, Alireza Fotuhi Siahpirani, Edwin M. Stone, Budd A. Tucker, , David M. Gamm

This study uses iPSC-RPE models to evaluate gene augmentation and editing for Best disease, finding that some mutations respond to gene therapy while others require genome editing.

bioRxiv · Jan 31, 2020 · PreprintGenetics