Author

Stephanie S. Steltzer

University of Michigan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Alireza Fotuhi SiahpiraniiD· 1 shared Amara Greer-ShortiD· 1 shared Amr A. AbdeeniD· 1 shared Ana BudaniD· 1 shared Anna Greenwood· 1 shared Benjamin SteyeriD· 1 shared Bikash Ranjan PattnaikiD· 1 shared Budd A. TuckeriD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperGenetics

Alireza Fotuhi SiahpiraniORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Hydrogel dimensionality directs mechanics-dependent nascent matrix deposition by tendon fibroblasts

Yu‐Chung Liu, Ziqian Yang, Stephanie S. Steltzer, Mingyue Fan, Evelyn Navarro Salazar, Jiahao Liang, Megan Leigh Killian, Claudia Loebel

This study shows that hydrogel dimensionality affects how tendon fibroblasts respond to mechanical cues, altering nascent matrix deposition.

bioRxiv · Oct 27, 2026 · PreprintBioengineering

Gene Therapy Mediates Therapeutic Improvement in Cardiac Hypertrophy and Survival in a Murine Model of MYBPC3-Associated Cardiomyopathy

Amara Greer-Short, Anna Greenwood, Elena C. Leon, Tawny Neal Qureshi, Konor von Kraut, Justin Wong, Jonathan H. Tsui, Christopher A. Reid, Ze Cheng, Emilee Easter, Jin Yang, Jaclyn J. Ho, Stephanie S. Steltzer, Ana Budan, Marie Cho, , Laura Marie Lombardi

This study demonstrates that AAV gene therapy with TN-201 restores MYBPC3 protein levels, reverses cardiac hypertrophy, and improves survival in a murine model of MYBPC3-associated HCM.

Nature Communications · Mar 4, 2025Peer-reviewedGenetics

Human iPSC modeling reveals mutation-specific responses to gene therapy in Best disease

Divya Sinha, Benjamin Steyer, Pawan Kumar Shahi, Katherine P. Mueller, Rasa Valiauga, Kimberly L. Edwards, Cole Bacig, Stephanie S. Steltzer, Sandhya Srinivasan, Amr A. Abdeen, Evan Cory, Viswesh Periyasamy, Alireza Fotuhi Siahpirani, Edwin M. Stone, Budd A. Tucker, , David M. Gamm

This study uses iPSC-RPE models to evaluate gene augmentation and editing for Best disease, finding that some mutations respond to gene therapy while others require genome editing.

bioRxiv · Jan 31, 2020 · PreprintGenetics