Author

Un In Chan

Weill Cornell MedicineORCID 0000-0001-6095-3565Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Christopher E. BarbieriiD· 2 shared Jude N. OwireduiD· 2 shared Alex Hakansson· 1 shared Andrea SboneriD· 1 shared Anuradha GopalaniD· 1 shared Anurag SharmaiD· 1 shared Brian D. RobinsoniD· 1 shared Chen Khuan WongiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCell Biology

Christopher E. BarbieriORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintFeb 16, 2026

Cell-Autonomous AR Dependence in Luminal Prostatic Epithelium Governs Survival and Lineage Plasticity

Dan Li, Naitao Wang, Wangxin Guo…·Yu Chen

This study shows that luminal prostatic epithelial cells depend on AR signaling for survival and differentiation, and their loss triggers compensatory pathways and lineage plasticity.

DOI 10.64898/2026.02.13.705807

mousebulk RNA-seq

𝕏 PostBluesky

Cancer BiologyNature CommunicationsPeer-reviewedDec 13, 2024

Canonical AREs are tumor suppressive regulatory elements in the prostate

Xuanrong Chen, Michael A. Augello, Deli Liu…·Christopher E. Barbieri

This study identifies canonical AREs as tumor suppressive regulatory elements in prostate cancer, showing their reactivation can induce death in AR+ cancer cells and is linked to better prognosis.

15 citations

humanbulk RNA-seq

𝕏 PostBluesky