Author

Katy L. Ong

University of California, BerkeleyORCID 0000-0001-6237-3580Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

David H. BilderiD· 2 shared Jan Mikhale B. CajulaoiD· 1 shared Jorian J. SepersiD· 1 shared Jung KimiD· 1 shared Karina Mai Anders· 1 shared Tsai‐Ching HsiiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 9 mutual co-author links

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2 shared papersCancer Biology

David H. BilderORCID

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2 papers

Recent·Most cited

A chronic interorgan wound response appropriated by Drosophila tumors to induce intestinal inflammation

Katy L. Ong, Jan Mikhale B. Cajulao, Karina Mai Anders, David H. Bilder

This paper shows Drosophila tumors exploit chronic wound healing pathways to disrupt intestinal homeostasis and induce inflammation through EGFR signaling.

bioRxiv · Jul 6, 2026 · PreprintCancer Biology

Systemic coagulopathy drives host lethality in a new Drosophila tumor model

Tsai‐Ching Hsi, Katy L. Ong, Jorian J. Sepers, Jung Kim, David H. Bilder

This study shows that a Drosophila tumor model induces coagulopathy phenotypes similar to human patients, with hypercoagulation followed by hypocoagulation, and demonstrates that rescuing coagulopathy improves survival.

bioRxiv · May 11, 2022 · PreprintDevelopmental Biology