Author

Sridhar Nonavinkere Srivatsan

Washington University in St. LouisORCID 0000-0001-6293-4632Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:long-read sequencing· 1

Frequent Co-Authors

Matthew J. WalteriD· 2 shared Amy M. Bertino· 1 shared Angelique Gilbert· 1 shared Casey D. S. KaterndahliD· 1 shared Catrina C. FronickiD· 1 shared Christopher A. MilleriD· 1 shared Claudia C. PastranaiD· 1 shared Kimmo PorkkaiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

Matthew J. WalterORCID

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2 papers

Recent·Most cited

Cancer BiologyLeukemiaPeer-reviewedOct 6, 2026

Sensitivity to ATR-CHK1 pathway inhibition in AML/MDS is enhanced by SRSF2 mutations and reduced by RUNX1 loss

Samuli Eldfors, Sumit Rai, Vineet Sharma…·Timothy Aaron Graubert

This study identifies SRSF2 and U2AF1 mutations as biomarkers of sensitivity to ATR-CHK1 pathway inhibitors and RUNX1 mutations as a mechanism of resistance in MDS/AML.

DOI 10.1038/s41375-026-03142-7

humanmouse

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Cancer BiologybioRxivPreprintJun 9, 2026

Long-read cDNA sequencing reveals novel isoforms and spliceosome-mutant-enriched transcripts in AML and MDS

Christopher A. Miller, Sridhar Nonavinkere Srivatsan, Michael H. Kramer…·Matthew J. Walter

This paper uses long-read cDNA sequencing to identify novel transcripts and spliceosome-mutant-enriched isoforms in AML and MDS, providing a resource for improving short-read RNA-seq analyses.

DOI 10.64898/2026.05.20.726635

humanlong-read sequencing

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