Jon F. Wilkins

This study shows that mice lacking paternal expression of Grb10 exhibit increased risk-taking behavior in specific tasks, supporting the idea that imprinted genes influence risk tolerance oppositely based on parental origin.

The paper shows that mice lacking paternal Grb10 make fewer impulsive choices, suggesting imprinted genes influence impulsive behavior and may contribute to pathological conditions.

The paper develops methods to infer generalized coalescent models and quantify uncertainty in their parameters using site-frequency spectra from Atlantic cod mitochondrial data.