Author

Xiao Yi Su

Rutgers, The State University of New JerseyORCID 0000-0001-6408-8273Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1

Frequent Co-Authors

Zhiping P. PangiD· 3 shared Le WangiD· 2 shared Anat KreimeriD· 1 shared Azadeh Jadali· 1 shared ChangHui PakiD· 1 shared Chao CheniD· 1 shared Christian HoffmanniD· 1 shared Chunyu LiuiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersNeuroscience

Zhiping P. PangORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cell BiologyScience AdvancesPeer-reviewedApr 18, 2025

Live-Cell Quantification Reveals Viscoelastic Regulation of Synapsin Condensates by α-Synuclein

Huan Wang, Christian Hoffmann, Johannes Vincent Tromm…·Zheng Shi

The paper quantifies viscoelastic properties of synapsin condensates in live cells and identifies α-synuclein's role in regulating their material state.

20 citationstop 1%

proteomicsexperimental assay

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NeurosciencebioRxivPreprintOct 20, 2024

Mutations of schizophrenia risk gene SETD1A dysregulate synaptic function in human neurons

Xiao Yi Su, Hanwen Zhang, Hong Yan…·Zhiping P. Pang

This study investigates how rare mutations in the SETD1A gene affect human neurons, showing they disrupt synaptic function and may contribute to schizophrenia pathophysiology.

1 citation

humanbulk RNA-seq

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NeuroscienceMolecular PsychiatryPeer-reviewedOct 24, 2022

Analyses of the Autism-associated Neuroligin-3 R451C Mutation in Human Neurons Reveals a Gain-of-Function Synaptic Mechanism

Le Wang, Vincent R. Mirabella, Rujia Dai…·Zhiping P. Pang

The paper investigates the impact of the NLGN3 R451C mutation on human neurons, finding it reduces protein levels but enhances excitatory synapses, potentially contributing to ASD pathogenesis.

53 citationstop 3%

humanscRNA-seq

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