Author

Connor S. Murphy

MaineHealthORCID 0000-0001-6533-3920Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Bree B. AldridgeiD· 1 shared Calvin P. H. VaryiD· 1 shared Carlos A. GartneriD· 1 shared Christian M. PottsiD· 1 shared Habib HamidiiD· 1 shared Heather E. FairfieldiD· 1 shared Joshua J. WhiteleyiD· 1 shared Kerri-Lynn Sheahan· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMicrobiology

Bree B. AldridgeORCID

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2 papers

Recent·Most cited

MicrobiologyScientific ReportsPeer-reviewedAug 8, 2026

Host CD59 Potentiates the Type III Secretion System in Yersinia pseudotuberculosis

Kristen J. Davis, Kerri-Lynn Sheahan, Connor S. Murphy…·Ralph R. Isberg

This study shows CD59 supports Yersinia pseudotuberculosis T3SS function by maintaining plasma membrane dynamics and lipid microdomain organization.

1 citation

experimental assayhost-pathogen & immune response

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Cancer BiologyMolecular OncologyPeer-reviewedJan 23, 2025

Inhibition of Acyl-CoA Synthetase Long Chain Isozymes Decreases Multiple Myeloma Cell Proliferation and Causes Mitochondrial Dysfunction

Connor S. Murphy, Heather E. Fairfield, Victoria E. DeMambro…·Michaela Ruth Reagan

This study shows that inhibiting ACSLs in multiple myeloma cells reduces proliferation and causes mitochondrial dysfunction, suggesting ACSLs as potential therapeutic targets.

18 citationstop 4%

humanbulk RNA-seq

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