Bruno Ramos‐Molina

This study identifies PTPRF as a stress-responsive cytoskeletal checkpoint that coordinates metabolic adaptation in hepatocytes and β cells by linking metabolic stress to actin reorganization.

This study shows that deleting HKDC1 in mice reduces Western diet-induced MASH by improving metabolism and reducing inflammation and fibrosis.

This study identifies increased NLRP1 expression in MASH liver samples and shows that palmitic acid activates the NLRP1 inflammasome in human cells.