Jonathan Underwood

This study uses dMRS to show IFN-β increases thalamic tCho diffusivity and age is linked to lower tNAA diffusivity and higher tCho/tCr in white matter.

This paper develops IFN-β as a safe in-vivo model of human inflammation, inducing robust sickness responses with minimal cardiovascular effects in healthy volunteers aged 18-75.

This study shows that Staphylococcus aureus infection increases NKG2A+ NK cells with higher IFN-γ production in humans, potentially driving inflammation and disease pathogenesis.