Neil Andrew Harrison

This study links human brain functional connectivity to neurotransmitter systems using resting-state fMRI and MEG across multiple cohorts, showing consistent patterns and clinical relevance.

This study identifies two brain connectivity patterns associated with breathlessness catastrophising in post-COVID patients, suggesting both interoceptive and visceromotor pathway alterations.

This study uses dMRS to show IFN-β increases thalamic tCho diffusivity and age is linked to lower tNAA diffusivity and higher tCho/tCr in white matter.

This paper develops IFN-β as a safe in-vivo model of human inflammation, inducing robust sickness responses with minimal cardiovascular effects in healthy volunteers aged 18-75.

This study shows that targeted memory reactivation during REM sleep reduces emotional responses by decreasing brain activity and physiological arousal to emotional stimuli.

The paper investigates the association between obesity and two brain systems using MRI data from UK Biobank participants, finding distinct structural and genetic relationships.

The paper examines sex differences in brain network development during adolescence and links them to depression risk, finding divergent patterns in functional connectivity and gene expression.

This study analyzes structural brain asymmetries in ADHD using 39 datasets, finding small effects that do not meet study-wide significance thresholds.

This study uses machine learning to show that structural brain anomalies in ADHD are similar in children and adults, supporting continuity of the disorder's pathophysiology.

This study uses REACT to analyze how methylphenidate affects dopaminergic and noradrenergic circuits in the brain and links these effects to behavioral responses in a reinforcement-learning task.

This study compares structural brain differences across ADHD, ASD, and OCD using MRI data from multiple cohorts, finding disorder-specific alterations in brain volume and cortical thickness.

This study finds elevated peripheral C-reactive protein (CRP) is associated with treatment-resistant depression, childhood adversity, and specific depressive and anxious symptoms.