Author

Vladimı́r Tesař

Charles UniversityORCID 0000-0001-6982-0689Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Bioinformatics· 1 Genetic & Genomic Medicine· 1 Genetics· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 3 Approach:statistical model· 3 Research Focus:GWAS / association mapping· 2 Assay:GWAS summary statistics· 2 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:optimal transport· 1

Frequent Co-Authors

Rosanna CoppoiD· 2 shared Adele MitrottiiD· 1 shared Aftab S. ChishtiiD· 1 shared Akchurin Oleh· 1 shared Alejandro Quiroga· 1 shared Andrea Magnano· 1 shared Andrew S. BombackiD· 1 shared Antonello Pani· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBioinformatics

Rosanna CoppoORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

BioinformaticsMolecular Systems BiologyPeer-reviewedDec 19, 2023

Detection of PatIent-Level distances from single cell genomics and pathomics data with Optimal Transport (PILOT)

Mehdi Joodaki, Mina Shaigan, Victor Parra…·Ivan G. Costa

PILOT uses optimal transport to analyze single-cell and pathomics data, detecting disease-associated samples and non-linear changes in cell populations and tissue structures.

34 citations

humanscRNA-seq

𝕏 PostBluesky

Genetic & Genomic MedicinemedRxivPreprintNov 19, 2021

GWAS defines pathogenic signaling pathways and prioritizes drug targets for IgA nephropathy

Krzysztof Kiryluk, Elena Sánchez, Shu‐Feng Zhou…·Claudia Izzi

This study identifies 30 genome-wide significant risk loci for IgA nephropathy, links them to inflammatory pathways, and prioritizes drug targets.

5 citations

humanGWAS summary statistics

𝕏 PostBluesky

GeneticsbioRxivPreprintDec 10, 2018

Genetically distinct clinical subsets, and associations with asthma and eosinophil abundance, within Eosinophilic Granulomatosis with Polyangiitis

Paul Lyons, James E. Peters, Federico Alberici…·Kenneth G. C. Smith

This study identifies genetic subsets of EGPA associated with asthma and eosinophil levels, revealing two distinct clinical and genetic syndromes.

10 citations

humanGWAS summary statistics

𝕏 PostBluesky