Richard A. Baylis

This study shows that CD4-CD8 T-cell interactions are necessary for cardiac injury, as perforin-dependent cytotoxicity drives tissue damage despite ongoing inflammation.

This study shows cancer increases atherosclerotic plaque vulnerability through pathological angiogenesis, and targets this pathway to prevent plaque destabilization.

This study identifies BST2 as a driver of vascular smooth muscle cell plasticity and macrophage-like phenotype switching in tumors, with implications for immunosuppression and therapy.