Author

Nicolas Gabriel Lopez

Stanford MedicineORCID 0000-0003-0659-4500Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Assay:scATAC-seq· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Clint L. MilleriD· 2 shared Nicholas James LeeperiD· 2 shared Wei FengiD· 2 shared Adam W. TurneriD· 1 shared Aloke Virmani FinniD· 1 shared Anshul Bharat KundajeiD· 1 shared Caitlin BelliD· 1 shared Changhao FuiD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

Clint L. MillerORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintSep 10, 2024

BST2 induces vascular smooth muscle cell plasticity and phenotype switching during cancer progression

Caitlin Bell, Richard A. Baylis, Nicolas Gabriel Lopez…·Nicholas James Leeper

This study identifies BST2 as a driver of vascular smooth muscle cell plasticity and macrophage-like phenotype switching in tumors, with implications for immunosuppression and therapy.

DOI 10.1101/2024.09.10.612298

mousescRNA-seq

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GenomicsNature GeneticsPeer-reviewedMay 19, 2022

Cell-specific chromatin landscape of human coronary artery resolves regulatory mechanisms of disease risk

Adam W. Turner, Shengen Shawn Hu, José Verdezoto Mosquera…·Clint L. Miller

The paper uses single-cell ATAC-seq to analyze coronary artery cells from CAD patients, identifying regulatory mechanisms and risk variants associated with disease.

159 citationstop 1%

humanscATAC-seq

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