Author

Lingfeng Luo

Stanford MedicineORCID 0000-0002-0601-0053Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Changhao FuiD· 2 shared Fudi WangiD· 2 shared Hua GaoiD· 2 shared Nicholas James LeeperiD· 2 shared Richard A. BaylisiD· 2 shared Shaunak Sanjay AdkariD· 2 shared Allen M. Haas· 1 shared Caitlin BelliD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersPathology

Changhao FuORCID

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2 papers

Recent·Most cited

Cancer drives atherosclerotic plaque vulnerability by inducing pathological angiogenesis

Lingfeng Luo, Changhao Fu, Kai-Uwe Jarr, Richard A. Baylis, Virginia H. Sun, Julius Cornelis Heemelaar, Moritz von Scheidt, Daniela Ramirez, Johannes Krefting, Nadja Sachs, Justus Leonard Wettich, Hanna Winter, Hua Gao, Fudi Wang, Shaunak Sanjay Adkar, , Nicholas James Leeper

This study shows cancer increases atherosclerotic plaque vulnerability through pathological angiogenesis, and targets this pathway to prevent plaque destabilization.

bioRxiv · Jan 20, 2026 · PreprintPathology

BST2 induces vascular smooth muscle cell plasticity and phenotype switching during cancer progression

Caitlin Bell, Richard A. Baylis, Nicolas Gabriel Lopez, Wei Feng, Hua Gao, Fudi Wang, Sharika Bamezai, Changhao Fu, Yoko Kojima, Shaunak Sanjay Adkar, Lingfeng Luo, Clint L. Miller, Nicholas James Leeper

This study identifies BST2 as a driver of vascular smooth muscle cell plasticity and macrophage-like phenotype switching in tumors, with implications for immunosuppression and therapy.

bioRxiv · Sep 10, 2024 · PreprintCancer Biology