Author

Nateka L. Jackson

University of Alabama at BirminghamORCID 0000-0001-7364-2440Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:rat· 2 Assay:electronic health records· 1

Frequent Co-Authors

Lori L. McMahoniD· 2 shared Abbi R. HernandeziD· 1 shared Anthoni M. GoodmaniD· 1 shared Bethany M. LangneriD· 1 shared Caesar M. HernandeziD· 1 shared Capri AlexiD· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 9 mutual co-author links

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2 shared papersNeuroscience

Lori L. McMahonORCID

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2 papers

Recent·Most cited

NeuroscienceeNeuroPeer-reviewedMay 1, 2022

Impairments in fear extinction memory and basolateral amygdala plasticity in the TgF344-AD rat model of Alzheimer's disease are distinct from non-pathological aging.

Caesar M. Hernandez, Nateka L. Jackson, Abbi R. Hernandez, Lori L. McMahon

This study investigates fear extinction memory and basolateral amygdala (BLA) synaptic dysfunction in TgF344-AD rats, finding distinct impairments in AD compared to non-pathological aging.

20 citations

ratelectronic health records

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NeuroscienceJournal of NeurosciencePeer-reviewedMay 5, 2021

Heightened β-adrenergic receptor function in the TgF344-AD rat model drives synaptic potentiation and supports learning and memory

Anthoni M. Goodman, Bethany M. Langner, Nateka L. Jackson…·Lori L. McMahon

The study finds heightened β-adrenergic receptor function in the TgF344-AD rat model enhances synaptic plasticity and preserves learning and memory in early AD.

64 citationstop 2%

ratexperimental assay

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