Author

Supriya Swarnkar

Scripps Research InstituteORCID 0000-0001-7596-0215Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1

Frequent Co-Authors

Neelam ShahaniiD· 2 shared Srinivasa SubramaniamiD· 2 shared Alessandro UsielloiD· 1 shared Catherina Scharager-TapiaiD· 1 shared Eddie GrinmaniD· 1 shared George TsaprailisiD· 1 shared Gogce C. CryneniD· 1 shared Isabel EspadasiD· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersNeuroscience

Neelam ShahaniORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintJun 16, 2021

Rhes Deletion Prevents Age-Dependent Selective Motor Deficits and Reduces Phosphorylation of S6K in Huntington Disease Hdh150Q(CAG) Knock-In Mice.

Jennifer Hernandez, Neelam Shahani, Supriya Swarnkar, Srinivasa Subramaniam

This study shows that Rhes deletion reduces age-dependent motor deficits and mTORC1-S6K hyperphosphorylation in a mouse model of Huntington disease.

3 citations

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintSep 10, 2020

A cAMP/PKA-dependent synaptically targeted lncRNA mediates structural plasticity in hippocampal neurons by functionally interacting with the Spectrin/Ankyrin Network

Eddie Grinman, Yoshihisa Nakahata, Yosef Avchalumov…·Sathyanarayanan V. Puthanveettil

The paper identifies an intronic lncRNA, ADEPTR, that is synaptically transported via cAMP/PKA and interacts with the Spectrin/Ankyrin network to regulate structural plasticity in hippocampal neurons.

1 citation

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 27, 2019

RasGRP1 (CalDAG-GEF-II) Mediates L-DOPA-induced Dyskinesia in a Mouse Model of Parkinson Disease

Mehdi Ishragi, Uri Nimrod Ramírez-Jarquín, Neelam Shahani…·Srinivasa Subramaniam

This study identifies RasGRP1 as a key mediator of L-DOPA-induced dyskinesia in a mouse model of Parkinson's disease, showing its role in multiple signaling pathways and potential as a therapeutic target.

1 citation

mouseexperimental assay

𝕏 PostBluesky