Author

Kui‐Jin Kim

Seoul National University Bundang HospitalORCID 0000-0001-7715-5121Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Eun Hee JungiD· 2 shared Jee Hyun KimiD· 2 shared Jin Won KimiD· 2 shared Koung Jin SuhiD· 2 shared Se Hyun KimiD· 2 shared Yu Jung KimiD· 2 shared Andrew J. AguirreiD· 1 shared Bo‐Ram ParkiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersCancer Biology

Eun Hee JungORCID

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2 papers

Recent·Most cited

Cancer BiologyCancer Gene TherapyPeer-reviewedOct 31, 2026

Loss of p21-activated kinase 4 (PAK4) suppresses pancreatic tumor progression and metastasis through regulating E-cadherin

Kui‐Jin Kim, Cheng Hyun Lee, Hee Young Na…·Jin Won Kim

This study shows that PAK4 promotes pancreatic cancer metastasis by reducing E-cadherin, and its loss inhibits tumor progression and metastasis.

DOI 10.1038/s41417-026-01091-y

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintSep 16, 2025

VEGFR2 blockade overcomes acquired KRAS G12D inhibitor resistance driven by PI3Kγ activation

Sung‐Hyun Hwang, Mingyun Bae, Ji‐Won Kim…·Keun‐Wook Lee

This paper identifies VEGFA-VEGFR2 signaling driven by PI3Kγ activation as a mechanism of acquired resistance to KRAS G12D inhibitors and shows that blocking this pathway restores drug sensitivity in resistant cancer models.

DOI 10.1101/2025.09.16.676456

𝕏 PostBluesky