Author

Jing Zeng

Shanghai Jiao Tong UniversityORCID 0000-0001-7764-155XUnclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Jing Zeng2 papers · Broad Institute

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Arun Jayant SanyaliD· 1 shared Derrick Zhao· 1 shared Dong LiuiD· 1 shared Emily C. Gurley· 1 shared Fei LiiD· 1 shared Giovanni TargheriD· 1 shared Grayson W. WayiD· 1 shared Huiping ZhouiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

Arun Jayant SanyalORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cell BiologyHepatology CommunicationsPeer-reviewedJul 28, 2025

Aramchol attenuates fibrosis in mouse models of biliary fibrosis and blocks the TGFβ-induced fibroinflammatory mediators in cholangiocytes

Sayed Obaidullah Aseem, Jing Wang, Maleeha F. Kalaiger…·Huiping Zhou

This study shows Aramchol reduces fibrosis in mouse models of biliary disease by inhibiting TGFβ-induced inflammation and fibrotic markers in cholangiocytes.

3 citations

mousebulk RNA-seq

𝕏 PostBluesky

Genetic & Genomic MedicinemedRxivPreprintFeb 3, 2023

Causal relationships between genetically predicted circulating levels of amino acids and non-alcoholic fatty liver disease risk: a Mendelian randomisation study

Jian Zhao, Jing Zeng, Dong Liu…·Jian‐Gao Fan

This study uses Mendelian randomisation to investigate the causal relationship between circulating amino acid levels and non-alcoholic fatty liver disease risk, finding higher alanine and lower glutamine levels are associated with increased and decreased risk, respectively.

1 citation

humanGWAS summary statistics

𝕏 PostBluesky