Author

Jasinda H. Lee

National University of SingaporeORCID 0000-0001-7779-8135Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genomics· 1 Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

Brian Keith KennedyiD· 2 shared Aarthi RavikrishnaniD· 1 shared Aeowynn J. CoakleyiD· 1 shared Ahmad Nazri Mohamed Naim· 1 shared Amanda Hui Qi NgiD· 1 shared Andrea Britta MaieriD· 1 shared Anis LarbiiD· 1 shared Anja KarlstaedtiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersMolecular Biology

Brian Keith KennedyORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Molecular BiologyCell MetabolismPeer-reviewedNov 25, 2025

A metabolic atlas of mouse aging

Steven E. Pilley, Dominik Awad, Djakim Latumalea…·Peter J. Mullen

This study maps metabolic changes in 12 organs of male and female mice across 5 ages, identifying sex-specific patterns and a metabolic aging clock.

25 citationstop 2%

mousemetabolomics

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GenomicsNature CommunicationsPeer-reviewedSep 5, 2024

Gut metagenomes of Asian octogenarians reveal metabolic potential expansion and distinct microbial species associated with aging phenotypes

Aarthi Ravikrishnan, Indrik Wijaya, Eileen Png…·Niranjan Nagarajan

The paper analyzes gut metagenomes of Asian octogenarians to identify age-related microbial shifts and their associations with aging phenotypes.

33 citationstop 3%

humanmetagenomics

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NeurosciencebioRxivPreprintApr 3, 2020

The lateral entorhinal cortex is a hub for local and global dysfunction in pre-tauopathy states

Francesca Mandino, Ling Yun Yeow, Renzhe Bi…·Joanes Grandjean

This study identifies a dichotomic hypo-connected resting and hyper-responsive active phenotype in a mouse model of early tauopathy, with implications for Alzheimer's disease.

1 citation

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