Author

Giulia Cesaro

University of PaduaORCID 0000-0001-7971-963XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genomics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1

Frequent Co-Authors

Hind MedyoufiD· 2 shared Ivan G. CostaiD· 2 shared Rafael KramanniD· 2 shared Rebekka K. SchneideriD· 2 shared Agnieszka Katarzyna MaciołaiD· 1 shared Aitor AguirreiD· 1 shared Alex A. ZhavoronkoviD· 1 shared Alfredo Pherez-FarahiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCancer Biology

Hind MedyoufORCID

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintOct 21, 2026

Sequential engagement of distinct dendritic cell subsets shapes Th2 polarization

Enrico Dotta, Nathan Sébastien Zangger, Giulia Cesaro…·Giulia Pasqual

The paper shows that distinct dendritic cell subsets sequentially engage CD4 T cells during priming, with late CD70 DC2s driving Th2 polarization.

DOI 10.64898/2026.10.15.759358

mouseexperimental assay

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Cancer BiologybioRxivPreprintMar 18, 2026

NicheSphere reveals Spp1⁺ macrophages as central hubs coordinating fibrotic remodeling in myeloproliferative neoplasms

Hélène F.E. Gleitz, Mayra L. Ruiz Tejada Segura, James Shiniti Nagai…·Rebekka K. Schneider

This study identifies Spp1⁺ macrophages as key hubs coordinating fibrotic remodeling in myeloproliferative neoplasms using NicheSphere, a novel computational method for cell communication analysis.

1 citation

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GenomicsbioRxivPreprintFeb 14, 2026

Polyploid cardiomyocytes define disease-specific transcriptional states in the mammalian heart

Paul Kießling, Mehdi Joodaki, Daryna Pikulska…·Christoph Kuppe

This study identifies polyploid cardiomyocytes as disease-specific transcriptional states in the mammalian heart and suggests targeting them for heart failure treatment.

5 citations

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