Sahel Jahangiri Esfahani

The paper identifies a paradoxical inflammation state in FM neutrophils that are unresponsive to LPS, linked to NF-κB suppression, and suggests bryostatin as a potential therapeutic target.

This study identifies GPR37L1 in satellite glial cells as a regulator of pain control through interactions with potassium channels and maresin 1, showing its role in neuropathic pain resolution.

The paper creates a database of pain-related transcriptomic data, identifies shared pathways, RNA biomarkers, and shows differential expression genes are more informative than tissue-specific genes in heritability analyses.