Author

Chisato Kodera

Institute of Molecular Embryology and Genetics, Kumamoto UniversityORCID 0000-0001-8091-0001Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Kei‐ichiro IshiguroiD· 2 shared Yuki Takada-HorisawaiD· 2 shared Akihiko SakashitaiD· 1 shared Atsushi SuzukiiD· 1 shared Hidetaka KatabuchiiD· 1 shared Hitoshi NiwaiD· 1 shared Kazumasa TakemotoiD· 1 shared Kenichi HorisawaiD· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersDevelopmental Biology

Kei‐ichiro IshiguroORCID

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2 papers

Recent·Most cited

Developmental BiologyPLoS GeneticsPeer-reviewedMay 7, 2021

KCTD19 associates with ZFP541 and HDAC1 and is required for meiotic exit in male mice

Seiya Oura, Takayuki Koyano, Chisato Kodera…·Masahito Ikawa

The paper identifies KCTD19 as essential for meiotic exit in male mice, showing it interacts with ZFP541 and HDAC1, and its deficiency causes apoptosis during meiosis.

34 citationstop 3%

mouseexperimental assay

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Developmental BiologybioRxivPreprintJan 15, 2021

Meiosis-specific ZFP541 repressor complex promotes meiotic prophase exit during spermatogenesis

Yuki Takada-Horisawa, Chisato Kodera, Kazumasa Takemoto…·Kei‐ichiro Ishiguro

This study identifies a ZFP541-KCTD19 repressor complex that promotes meiotic prophase exit in spermatogenesis by suppressing transcriptional regulators and chromatin modifiers.

1 citation

mouseChIP-seq

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