Author

Maykelis Díaz

University of MiamiORCID 0000-0001-8149-2948Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:ATAC-seq· 1 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Assay:electrophysiology· 1 Approach:experimental assay· 1

Frequent Co-Authors

René Barro-SoriaiD· 2 shared Alicia de la CruziD· 1 shared Andy Hinojo-PereziD· 1 shared Carsten MimiD· 1 shared Derek M. DykxhoorniD· 1 shared Gerhard DahliD· 1 shared Iqra Shams· 1 shared Junjie WangiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBiophysics

René Barro-SoriaORCID

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2 papers

Recent·Most cited

BiophysicsFEBS JournalPeer-reviewedMar 14, 2025

A metastasis-associated Pannexin1 mutant (Panx11-89) forms a minimalist ATP release channel

Junjie Wang, Noah J. Levi, Maykelis Díaz…·René Barro-Soria

This study shows that the Panx11-89 mutant forms a functional ATP release channel independently of wild-type Panx1, with conserved structure-function features and sensitivity to inhibitors.

5 citations

humanATAC-seq

𝕏 PostBluesky

BiophysicsCommunications BiologyPeer-reviewedSep 19, 2024

Lipophilic compounds restore wt function of neurodevelopmental-associated KCNQ3 mutations.

Michaela A. Edmond, Andy Hinojo-Perez, Mekedlawit Efrem…·René Barro-Soria

The paper investigates how two KCNQ3 mutations impair channel function and shows that polyunsaturated fatty acids can restore wild-type function in these mutations.

5 citations

humanelectrophysiology

𝕏 PostBluesky